Resources
Citable Research
- Lack of evidence for the effectiveness or safety of over-the-counter cannabidiol products
- The endocannabinoid system: an emerging key player in inflammation
- The Endocannabinoid System and Anxiety
- Cannabidiol as an emergent therapeutic strategy for lessening the impact of inflammation on oxidative stress
- Cannabinoids suppress inflammatory and neuropathic pain by targeting α3 glycine receptors
- Cannabidiol reduces intestinal inflammation through the control of neuroimmune axis
- Cannabidiol attenuates cisplatin-induced nephrotoxicity by decreasing oxidative/nitrosative stress, inflammation, and cell death
- Cannabinoids, inflammation, and fibrosis
- Cannabinoids for neuropathic pain
- The non-psychoactive cannabis constituent cannabidiol is an orally effective therapeutic agent in rat chronic inflammatory and neuropathic pain
- Cannabidiol displays anti-epileptiform and anti-seizure properties in vitro and in vivo
- Role of the cannabinoid system in pain control and therapeutic implications for the management of acute and chronic pain episodes
- CBD as an anxiolytic drug
- CBD reduces the anxiety induced by simulated public speaking in treatment-naïve social phobia patients
- Antidepressant-Like and Anxiolytic-Like Effects of Cannabidiol: A Chemical Compound of Cannabis Sativa
- CBD regulation of emotion and emotional memory processing: relevance for treating anxiety-related and substance abuse disorders
- Cannabinoids provide neuroprotection against 6-hydroxydopamine toxicity in vivo and in vitro: relevance to Parkinson’s disease
- Effectiveness of Cannabidiol Oil for Pediatric Anxiety and Insomnia as Part of Posttraumatic Stress Disorder: A Case Report.
- Endocannabinoid Signaling in Autism
- Cannabidiol Based Medical Cannabis in Children with Autism—A Retrospective Feasibility Study
“Recent studies suggest that cannabidiol may have utility in treating a number of human diseases and disorders now known to involve activation of the immune system and associated oxidative stress, as a contributor to their etiology and progression. These include rheumatoid arthritis, types 1 and 2 diabetes, atherosclerosis, Alzheimer disease, hypertension, the metabolic syndrome, ischemia-reperfusion injury, depression, and neuropathic pain.”
—Department of Pharmacology and Toxicology, School of Medicine, and Center for Excellence in Cardiovascular-Renal Research, University of Mississippi Medical Center
